Posted by Richard Willet - Memes and headline comments by David Icke Posted on 15 November 2022

Study: 100% Of mRNA Jab Recipients Have Heart Implications

Reluctance to Publicly Address Post-Jab Long COVID

In January 2021, National Institutes of Health researchers initiated testing and attempted treatment of patients suspected of having long COVID following their shot, but for unknown reasons the investigation petered out by the end of the year, leaving patients high and dry, without answers.5

According to Science, NIH researchers did continue their work “behind the scenes,” and other researchers, worldwide, have also started studying the phenomenon. Still, there appears to be extreme reluctance to addressing post-jab long COVID symptoms publicly. Why?

Dr. Avindra Nath, clinical director at the National Institute of Neurological Disorders and Stroke (NINDS) and the one leading the NIH’s investigation into long COVID, gives us a clue.

“Probing possible side effects presents a dilemma to researchers: They risk fomenting rejection of vaccines that are generally safe, effective, and crucial to saving lives,” Science writes.6 “‘You have to be very careful’ before tying COVID-19 vaccines to complications, Nath cautions. ‘You can make the wrong conclusion … The implications are huge.’”

In other words, it’s all about protecting the vaccine industry, which has now merged with and become the experimental gene therapy industry.

Meanwhile, the human test subjects are left to suffer — many of whom don’t even realize that they ARE test subjects. They bought the “safe and effective” and “rigorously tested” lies. In Nath’s defense, he tried to publish a case series on about 30 of these patients but medical journals refused to publish it.7

What’s Causing Long COVID?

As for the mechanisms behind long COVID, opinions vary. Research8,9 presented10 by Dr. Bruce Patterson at the International COVID Summit in Rome, in September 2021, suggests monocytes, shown to cause lung damage in patients with acute COVID, are also involved in long COVID.

In summary, the inflammatory cytokines that are supposed to trigger T cell activation fail to do so in some people, resulting in an inadequate antiviral response. Instead of T cells — which are needed to quell the infection — B cells and a particular subset of monocytes are elevated. As described by

“When they used antibodies to look for evidence of coronavirus proteins in the monocytes … they found them — in spades. Seventy-three percent of the ‘non-classical’ monocytes in long-COVID patients carried the coronavirus proteins …

These types of monocytes have often been thought to be anti-inflammatory, but recent studies show that they can, in some situations, produce pro-inflammatory cytokines. They’re mostly involved in ‘trash cleanup,’ and the antiviral response …

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